Pharmacodynamics is the study of a drug's molecular, biochemical, and physiologic effects or actions. It comes from the Greek words "pharmakon" meaning "drug" and "dynamikos" meaning "power." All drugs produce their effects by interacting with biological structures or targets at the molecular level to induce a change in how the target molecule functions in regards to subsequent intermolecular interactions. These interactions include receptor binding, post-receptor effects, and chemical interactions. Examples of these types of interactions include drugs binding to an active site of an enzyme, drugs that interact with cell surface signaling proteins to disrupt downstream signaling, and drugs that act by binding molecules like tumor necrosis factor (TNF).Subsequent to the drug-target interaction occurring downstream, effects are elicited which can be measured by biochemical or clinical means. Examples of this include the inhibition of platelet aggregation after administering aspirin, the reduction of blood pressure after ACE inhibitors, and the blood-glucose-lowering effect of insulin. While these examples seem obvious, the administration of the preceding drug examples should be kept in mind so practitioners do not administer these drugs to inhibit platelet aggregation, lower blood pressure or lower blood glucose but to reduce the risks of cerebrovascular accident, myocardial infarction, and renal and eye complications through the drug's pharmacodynamic effects

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With Regards,

Anna D Parker

Editorial Assistant

Journal of pharmaceutica Analytica Acta

Email: pharmaanalytica@engjournals.com